Mark recently posted a really good interview with Dr. Pim van Lommel:
After the interview, Mark followed up with him about the latest sketchy anti-NDE science piece that's making the rounds:
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Mark Ireland wrote:
Hi Alex, I’m not sure if you read either of these articles (on the same topic) but they seem to be another attempt at a materialist explanation for NDE’s.
https://www.smithsonianmag.com/smar...-flashes-before-our-eyes-upon-death-180979647
https://bgr.com/science/the-brain-activity-of-a-dying-person-was-recorded-for-the-first-time-ever
I wanted to get ahead of this with the members of Helping Parents Heal, so I wrote to Pim Van Lommel, asking for his response, as I felt the articles were being “sneaky” with some of their wording and specifics. To my surprise, Dr. Van Lommel provided an extremely thorough response, and wanted to share it with you in case this topic comes up with your listeners.
See his response below—and he also sent three attachments.
Mark Ireland
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From: Pim van Lommel
To: 'Mark Ireland'
Sent: Friday, March 4, 2022, 02:36:08 AM PST
Dear Mark,
There have been a lot of questions from all over the world about this article, which had a lot of attention in the popular press.
In this article the brain activity is described in a dying patient with already severe brain damage. The authors describe a short increase in gamma waves at the moment that according to their criteria the patient has a cardiac arrest. They define the cardiac arrest as ‘the abrupt loss of heart function measured by the inability to obtain pulse activity in the EKG’. They write that the patient developed a ventricular tachycardia with apneustic respirations and a clinical cardiorespiratory arrest. However, in figure 2A still electrical activity in the EKG is monitored beyond the moment that in the figure is described that the patient has a cardiac arrest!
According to worldwide accepted cardiological criteria: a cardiac arrest in a patient with an acute myocardial infarction is caused by ventricular fibrillation (VF) or by asystole (a flatline on the EKG). This is called ‘clinical death’, and this period of unconsciousness is reversible if adequate CPR with defibrillation or pacemaker is initiated within 5 – 10 minutes after the onset of cardiac arrest. If not, all patients with cardiac arrest will die due to irreversible damage to the brain. Clinical death is the first stage of the process of dying. A cardiac arrest by VF causes anoxia of the brain, which is a total lack of oxygen (no flow).
However, the patient in this article was shown to have a ventricular tachycardia (not VF or asystole). And the patient developed a very low bloodpressure by this ventricular tachycardia. In this situation there is often no a palpable pulse, in combination with the loss of consciousness, but in the brain there is hypoxia, a lack of oxygen (low flow), and not anoxia as in VF. When no CPR is initiated (electical cardioversion) the ventricular tachycardia will after some time change into VF, and the patient will die.
The authors write that an increase in gamma waves in the EEG is involved in cognitive processes and memory recall in healthy subjects, and that it is intriguing to speculate that such activity could support a last ‘recall’ that may take place in the near-death state.
However, they also write that there are seven important caveats that must be considered before generalizing these findings to understand the typical patterns of brain activity during death.
These caveats are 1) the fact that the described patient had a posttraumatic brain that suffered from hemorrhage, swelling and seizures, 2) anesthesia-induced loss of consciousness can alter neuronal oscillations, 3) dissociative events and drugs can cause an increase in gamma activity, 4) the patient had been placed on significant doses of anticonvulsant medication, which could directly affect the neuronal network activity, 5) asphyxia and hypercapnia can enhance cortical connectivity, and in this patient hypercapnia and resulting acidosis may have stimulated gap-junction activity, that is critical for gamma oscillations, 6) in this patient no normal activity was recorded in the EEG that can serve as a true baseline for comparison, 7) stereotyped neuronal activity patterns are conserved during daily behavioral tasks, but systematic research about brain activity during the process of dying has never been done. See my detailed comments and quotes in the attachment.
The authors write: We do not anticipate death in healthy subjects (NB. Van Lommel : like in patients in acute cardiac arrest during myocardial infarction) and therefore could not obtain uninterrupted recordings in the near-death phase in anything other than from circumstances involving pathological conditions in acute care hospital settings.
It is well established what happens in the EEG in a patient with an acute cardiac arrest, and who was healthy until that moment. The EEG changes after about 8 seconds after the onset of cardiac arrest, and becomes a flatline EEG after 18 seconds. See the attachment with some paragraphs from my latest article ‘The continuity of Consciousness’.
Summarizing: What the ‘general press’ writes about the content and conclusions of the already frequently quoted article is incomplete and mostly wrong, and seems to be used to find a materialist explanation for the cause of an NDE.
There have been some very critical comments about the interpretation of the EEG registrations in this article as well.
A commentary will follow in the JNDS.
Kind regards,
Pim van Lommel